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Aneurysm of the left atrium in a child with patent ductus arteriosus and mitral valve prolapse

Danielle Lilia Dantas TukamotoI; Carlos Henrique De MarchiI; Lilian BeaniI; Ulisses Alexandre CrotiI

DOI: 10.5935/1678-9741.20120082


bat/min: Batimentos por minuto

PCA: Persistência de canal arterial


Infant 9 months, 6.5 kg, female with history of dyspnea during feeding and poor weight gain. Born at term weighing 3.2 kg. In the first month, was diagnosed with congenital heart disease with increased pulmonary hemodynamic, being the child sent from her hometown for surgical treatment.

Physical examination showed malnutrition with weight below the percentile 3 for age, tachydyspneic, acyanotic and pale. Hyperdynamic precordium with apical impulse deviated to the left and chest bulging to the left. Continuous murmur 3 + / 6 +, with multiple clicks in the left infraclavicular region. Liver 3 cm from the right costal margin. Large and palpable peripheral pulses in all four limbs.



Sinus tachycardia with frequency of 150 beats / min, SAQRS + 60 º, and atrial and left ventricular (Figure 1).




Enlarged cardiac area, cardiothoracic ratio of 0.61, an increase of left atrium and left ventricle. Increased pulmonary vascular network (Figure 2).




Patent ductus arteriosus (PDA) with 7.09-mm and 4.48 mm aortic end in the lung. Mitral valve prolapse with mild to moderate degree. Dilation of the left atrium and moderate dilation of the left ventricle. Preserved ventricular function (Figure 3).




The clinical condition was compatible with the acyanogenic congenital heart disease with increased pulmonary blood flow, but not so usual effects of PDA in volume overload of the left chambers were potentiated by the presence of mitral valve prolapse with significant regurgitation. There is a clear indication of late immediate surgical treatment with echocardiographic monitoring aspect of the left atrium and mitral valve function [1,2].



During monitoring, it was possible to observe the clinical symptoms, with significant bulging chest to the left (Figure 4). The operation was performed routinely by left posterolateral thoracotomy, small incision, opening the parietal pleura, dissection of the PDA region close to the aorta, identification of anatomical structures, passing two 4-0 polypropylene threads around the canal, being one at the aortic end and the other at the pulmonary end and strongly bound, interrupting the blood flow passage. Suture of the parietal pleura, chest tube and chest closure performed by planes.



The child progressed well and was discharged on the third postoperative day and the echocardiogram revealed significant reduction of mitral valve prolapse and trivial regurgitation.


1. Zonnenberg I, de Wall K. The definition of a haemodynamic significant duct in randomized controlled trials: a systematic literature review. Acta Paediatr. 2012;101(3):247-51. [MedLine]

2. Schneider DJ, Moore JW. Patent ductus arteriosus. Circulation. 2006;114(17):1873-82. [MedLine]

Article receive on Friday, August 10, 2012

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